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Andrea Cignarella

Researcher at University of Padua

Publications -  92
Citations -  3090

Andrea Cignarella is an academic researcher from University of Padua. The author has contributed to research in topics: Estrogen & Estrogen receptor. The author has an hindex of 30, co-authored 85 publications receiving 2636 citations. Previous affiliations of Andrea Cignarella include University of Münster & University of Parma.

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Gender Differences in Endothelial Progenitor Cells and Cardiovascular Risk Profile The Role of Female Estrogens

TL;DR: EPCs are mobilized cyclically in fertile women, likely to provide a pool of cells for endometrial homeostasis and could represent one mechanism of protection in the fertile female population.
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Convenience versus Biological Significance: Are PMA-Differentiated THP-1 Cells a Reliable Substitute for Blood-Derived Macrophages When Studying in Vitro Polarization?

TL;DR: It is suggested that THP-1 be regarded as a simplified model of human macrophages when investigating relatively straightforward biological processes, such as polarization and its functional implications, but not as an alternative source in more comprehensive immunopharmacology and drug screening programs.
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Metabolic syndrome, inflammation and atherosclerosis.

TL;DR: The inflammatory component of atherogenesis has been increasingly recognized over the last decade and emerging therapies offer potential as future therapeutic approaches in preventing cardiovascular disease and, potentially, diabetes.
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Novel lipid-lowering properties of Vaccinium myrtillus L. leaves, a traditional antidiabetic treatment, in several models of rat dyslipidaemia: a comparison with ciprofibrate.

TL;DR: Findings indicate that active consituent(s) of Vaccinium myrtillus L. (blueberry) leaves may prove potentially useful for treatment of dyslipidaemiae associated with impaired TG-rich lipoprotein clearance.
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Rupture of the Atherosclerotic Plaque Does a Good Animal Model Exist

TL;DR: Attention should focus on the processes of plaque breakdown and thrombus formation in humans, whereas the use of animal models should probably be reserved for studying the function of particular genes and for investigating isolated features of plaques, such as the relationship between cap thickness and plaque stability.