M
Markku O. Pentikäinen
Researcher at University of Helsinki
Publications - 53
Citations - 3943
Markku O. Pentikäinen is an academic researcher from University of Helsinki. The author has contributed to research in topics: Low-density lipoprotein & Complement system. The author has an hindex of 32, co-authored 53 publications receiving 3616 citations. Previous affiliations of Markku O. Pentikäinen include Helsinki University Central Hospital & Forest Research Institute.
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Percutaneous coronary angioplasty versus coronary artery bypass grafting in treatment of unprotected left main stenosis (NOBLE): a prospective, randomised, open-label, non-inferiority trial.
Timo H. Mäkikallio,Niels Ramsing Holm,Mitchell Lindsay,Mark S. Spence,Andrejs Erglis,Ian B. A. Menown,Thor Trovik,Markku Eskola,Hannu Romppanen,Thomas Kellerth,Jan Ravkilde,Lisette Okkels Jensen,Gintaras Kalinauskas,Rikard Linder,Markku O. Pentikäinen,Anders Hervold,Adrian P. Banning,Azfar Zaman,Jamen Cotton,Erlend Eriksen,Sulev Margus,Henrik Toft Sørensen,Henrik Toft Sørensen,Per Hostrup Nielsen,Matti Niemelä,Kari Kervinen,Jens Flensted Lassen,Michael Maeng,Keith G. Oldroyd,Geoff Berg,Simon J Walsh,Colm G. Hanratty,Indulis Kumsars,Peteris Stradins,Terje K. Steigen,Ole Fröbert,Alastair N J Graham,Petter C Endresen,Matthias Corbascio,Olli A. Kajander,Uday Trivedi,Juha Hartikainen,Vesa Anttila,David Hildick-Smith,Leif Thuesen,Evald Høj Christiansen +45 more
TL;DR: In this article, the authors compared percutaneous coronary intervention (PCI) and coronary artery bypass grafting (CABG) for the treatment of left main coronary artery disease.
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Structure of low density lipoprotein (LDL) particles: basis for understanding molecular changes in modified LDL.
TL;DR: Structural information is utilized to understand and explain the molecular characteristics and interactions of modified, atherogenic LDL particles.
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Aggregation, fusion, and vesicle formation of modified low density lipoprotein particles: molecular mechanisms and effects on matrix interactions.
TL;DR: Molecular mechanisms that provide clues as to how aggregated lipid droplets and vesicles may be derived from modified LDL particles are discussed and how these modified forms of LDL, by means of their trapping to the extracellular matrix, may lead toextracellular lipid accumulation in the arterial intima.
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Modified LDL – trigger of atherosclerosis and inflammation in the arterial intima
TL;DR: Modified LDL – trigger of atherosclerosis and inflammation in the arterial intima (Minisymposium).
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TNFα Down-Regulates the Fas Ligand and Inhibits Germ Cell Apoptosis in the Human Testis
Virve Pentikäinen,Krista Erkkilä,Laura Suomalainen,Marjut Otala,Markku O. Pentikäinen,Martti Parvinen,Leo Dunkel +6 more
TL;DR: It is found that TNFalpha effectively and dose-dependently inhibited germ cell apoptosis, which was induced in vitro by incubating segments of human seminiferous tubules under serum-free culture conditions.