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Andrea Cossarizza

Researcher at University of Modena and Reggio Emilia

Publications -  487
Citations -  34586

Andrea Cossarizza is an academic researcher from University of Modena and Reggio Emilia. The author has contributed to research in topics: Immune system & Mitochondrion. The author has an hindex of 78, co-authored 448 publications receiving 30168 citations. Previous affiliations of Andrea Cossarizza include Academy for Urban School Leadership & University of Urbino.

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Decreased Circulating mtDNA Levels in Professional Male Volleyball Players.

TL;DR: Regular physical activity appeared associated with lower levels of circulating mtDNA, further confirming the protective, anti-inflammatory effect of exercise.
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Development of real time PCR assays for the quantification of Fas and FasL mRNA levels in lymphocytes: studies on centenarians.

TL;DR: The preliminary results suggest that during aging a subtle balance in the production of molecules that cause apoptosis could exist, and that, in order to avoid an excessive death of immune cells, a still unknown mechanism could compensate the increase of Fas with the reduction of FasL.
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Effect of antioxidants on mitochondrial function in HIV-1-related lipoatrophy: a pilot study.

TL;DR: It is shown that antioxidant supplementation may have a protective role on mitochondrial function, with limited effects on the reversal of clinical lipodystrophic abnormalities in HIV-1-infected patients.
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Kinetics of CD4 cells after discontinuation of antiretroviral therapy in patients with virological failure and a CD4 cell count greater than 500 cells/μl

TL;DR: After a median of 37 months on antiretroviral therapy, 16 patients were asked to discontinue treatment instead of changing it, and most patients experienced a rapid and progressive decrease in their CD4 cell count, even without a high viral load rebound.
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Mitochondrial functionality and metabolism in T cells from progressive multiple sclerosis patients

TL;DR: The data suggest that profound differences exist in the phenotypic and metabolic features of T cells from PP and SP patients, even though the two clinical phenotypes are considered part of the same disease spectrum.