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Andrea Cossarizza
Researcher at University of Modena and Reggio Emilia
Publications - 487
Citations - 34586
Andrea Cossarizza is an academic researcher from University of Modena and Reggio Emilia. The author has contributed to research in topics: Immune system & Mitochondrion. The author has an hindex of 78, co-authored 448 publications receiving 30168 citations. Previous affiliations of Andrea Cossarizza include Academy for Urban School Leadership & University of Urbino.
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Journal ArticleDOI
Autogeneic but Not Allogeneic Earthworm Effector Coelomocytes Kill the Mammalian Tumor Cell Target K562
Edwin L. Cooper,Andrea Cossarizza,Michael M. Suzuki,Stefano Salvioli,Miriam Capri,Daniela Quaglino,Claudio Franceschi +6 more
TL;DR: Recognition of, binding to, and killing of foreign cells in a natural killer cell-like reaction may reflect natural immunity in earthworms.
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Spontaneous and mitomycin-C-induced micronuclei in human lymphocytes exposed to extremely low frequency pulsed magnetic fields.
Maria Rosaria Scarfì,Ferdinando Bersani,Andrea Cossarizza,Daniela Monti,Gastone Castellani,Ruggero Cadossi,G. Franceschetti,Claudio Franceschi +7 more
TL;DR: Exposure to pulsed magnetic fields did not affect the frequency of micronuclei induced by mitomycin-C, suggesting that, in the experimental conditions used, this kind of field neither affected the integrity of chromosomes nor interfered with the genotoxic activity of mitomyin-C.
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Inhibition of Lon protease by triterpenoids alters mitochondria and is associated to cell death in human cancer cells.
Lara Gibellini,Marcello Pinti,Regina Bartolomeo,Sara De Biasi,Antonella Cormio,Clara Musicco,Gianluca Carnevale,Simone Pecorini,Milena Nasi,Anto De Pol,Andrea Cossarizza +10 more
TL;DR: The overexpression of Lon can rescue cells from cell death, providing an additional evidence on the role of Lon in conditions of excessive stress load.
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Persistent inflammation in HIV infection: established concepts, new perspectives.
TL;DR: New mechanisms were identified, so opening new perspective on the innate immune sensing in HIV-1 infection, and also different types of HIV-related nucleic acids can act as potent stimulators of innate immunity.
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Silencing of mitochondrial Lon protease deeply impairs mitochondrial proteome and function in colon cancer cells
Lara Gibellini,Marcello Pinti,Federica Boraldi,Valentina Giorgio,Paolo Bernardi,Regina Bartolomeo,Milena Nasi,Sara De Biasi,Sonia Missiroli,Gianluca Carnevale,Lorena Losi,Anna Tesei,Paolo Pinton,Daniela Quaglino,Andrea Cossarizza +14 more
TL;DR: Lon is essential for maintaining mitochondrial shape and function, and for survival of RKO cells and the triterpenoid 2‐cyano‐3,12‐dioxooleana‐1‐9,‐dien‐28‐oic acid, a Lon inhibitor, partially mimics Lon silencing.