scispace - formally typeset
A

Andrew Charles

Researcher at University of California, Los Angeles

Publications -  126
Citations -  18008

Andrew Charles is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Migraine & Cortical spreading depression. The author has an hindex of 53, co-authored 121 publications receiving 16197 citations. Previous affiliations of Andrew Charles include Mayo Clinic.

Papers
More filters
Journal ArticleDOI

The International Classification of Headache Disorders, 3rd edition (beta version)

Jes Olesen, +131 more
- 01 Jul 2013 - 
TL;DR: The International Classification of Headache Disorders, 3 edition (beta version), may be reproduced freely for scientific, educational or clinical uses by institutions, societies or individuals as mentioned in this paper. But the authors require the permission of the International Headache Society.
Journal ArticleDOI

Intercellular calcium signaling in astrocytes via ATP release through connexin hemichannels.

TL;DR: Release of ATP through connexin hemichannel activator quinine represents a novel signaling pathway for intercellular communication in astrocytes and other non-excitable cells.
Journal ArticleDOI

ATP Released from Astrocytes Mediates Glial Calcium Waves

TL;DR: The present experiments identify ATP as a major extracellular messenger in this system and implicate ATP as an important transmitter between CNS astrocytes.
Journal ArticleDOI

Intercellular signaling in glial cells: Calcium waves and oscillations in response to mechanical stimulation and glutamate

TL;DR: Intercellular Ca2+ signaling in primary cultures of glial cells was investigated with digital fluorescence video imaging and suggests distinct mechanisms for intra- and inter cellular Ca 2+ signaling may coordinate cooperative glial function.
Journal ArticleDOI

bcl-2 inhibits death of central neural cells induced by multiple agents.

TL;DR: It is reported that bcl-2 inhibits the death of a central neural cell line due to serum and growth factor withdrawal, the calcium ionophore A23187, glucose withdrawal, membrane peroxidation, and, in some cases, free radical-induced damage.