A
Andrew Metha
Researcher at University of Melbourne
Publications - 79
Citations - 2768
Andrew Metha is an academic researcher from University of Melbourne. The author has contributed to research in topics: Adaptive optics & Spatial frequency. The author has an hindex of 25, co-authored 74 publications receiving 2545 citations. Previous affiliations of Andrew Metha include McGill University & Australian National University.
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Rapid Adaptation in Visual Cortex to the Structure of Images
TL;DR: The authors showed that complex cells in striate cortex of macaque showed a rapid pattern-specific adaptation, which reduced correlations among the responses of populations of cells, thereby increasing the information transmitted by each action potential.
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Packing arrangement of the three cone classes in primate retina.
TL;DR: A detailed analysis of the spatial arrangement of L, M and S cones in the living eyes of two humans and one monkey using statistical methods that characterize the arrangement of each type of cone in the mosaic of photoreceptors.
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Functional consequences of the relative numbers of L and M cones
David H. Brainard,Austin Roorda,Yasuki Yamauchi,Jack B. Calderone,Andrew Metha,Maureen Neitz,Jay Neitz,David R. Williams,Gerald H. Jacobs +8 more
TL;DR: This study examines the functional consequences of variation in the relative numbers of L and M cones (L/M cone ratio) for two observers whose ratios were measured by direct imaging and indicates that neural factors play an important role in stabilizing unique yellow against variation inThe L/M cones ratio.
Journal Article
Functional consequences of the relative numbers of L and M cones (vol 17, pg 607, 2000)
David H. Brainard,Austin Roorda,Yasuki Yamauchi,Jack B. Calderone,Andrew Metha,Maureen Neitz,Jay Neitz,Williams,Gerald H. Jacobs +8 more
Journal ArticleDOI
Information Conveyed by Onset Transients in Responses of Striate Cortical Neurons
TL;DR: The responses of neurons in striate cortex to stationary grating patterns presented with abrupt onset are characterized, which are distinctive and well captured by a model in which each excitatory synaptic event leads to an immediate reduction in synaptic gain, from which recovery is slow.