Angelika Schobert

TL;DR: Primary rat astrocyte cultures were used to isolate a macrophage population that does not adhere to the confluent glial cells and characterized them as a distinct cell population that shares features both of peritoneal macrophages and of astroglial cells.

TL;DR: The present results identify one adenyl compound thus released, namely ATP, and identify astrocytes as one source and the release is brought about by activation of any of the three ionotropic glutamate receptor types-N-methyl-D-aspartate, AMPA and kainate receptors.

TL;DR: The results demonstrate directly the action potential-evoked, Ca(2+)-dependent and presumably vesicular and exocytotic release of ATP from postganglionic sympathetic neurons that support the co-transmitter theory and suggest that cultured sympathetic neurons are a preparation in which noradrenaline-ATP co-release can be examined free from postjunctional components.

TL;DR: It is concluded that IL-3 expression in ensuring the recruitment of enhanced numbers of immunocompetent cells at sites of lesion is a partial compensation of T cell functions in brain lesions.

TL;DR: Evidence of P2-purinoceptors on cultured astroglial cells is given and it is shown that pertussis toxin sensitive G-proteins influence some early step in prostaglandin synthesis.