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Antonio Adilton Oliveira Carneiro

Researcher at University of São Paulo

Publications -  105
Citations -  1425

Antonio Adilton Oliveira Carneiro is an academic researcher from University of São Paulo. The author has contributed to research in topics: Imaging phantom & Transducer. The author has an hindex of 20, co-authored 102 publications receiving 1215 citations. Previous affiliations of Antonio Adilton Oliveira Carneiro include Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto.

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Focused ultrasound neuromodulation of cortical and subcortical brain structures using 1.9 MHz

TL;DR: The capability of focused ultrasound (FUS) neuromodulation in the megahertz-range to achieve superior targeting specificity in the murine brain as well as demonstrate modulation of both motor and sensory responses demonstrates the capability of FUS to perform functional brain mapping.
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Nonlinear elastic behavior of phantom materials for elastography

TL;DR: The insights provided by this study will form the basis for stable elastography phantoms with stiffness and nonlinear stress/strain relationships in the background that differ from those in the target.
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Paraffin-Gel Tissue-Mimicking Material for Ultrasound-Guided Needle Biopsy Phantom

TL;DR: Results indicate that paraffin-gel waxes may be promising materials for training radiologists in ultrasound biopsy procedures and possesses long-term stability and can be employed in a supine position without changes in geometry.
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MRI relaxometry: methods and applications

TL;DR: In this article, the influence of pulse sequences and parameters on transversal relaxation time (T2) measurements in human tissues was analyzed for different tissues, and the effect of repetition and echo time, predicted in sequences of signal acquisition, was evaluated using simulated MRI signal.
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Evaluation of Delayed Neuronal and Axonal Damage Secondary to Moderate and Severe Traumatic Brain Injury Using Quantitative MR Imaging Techniques

TL;DR: These quantitative MR imaging techniques could noninvasively demonstrate the neuronal and axonal damage in the NAWM and CC of human brains, secondary to moderate or severe TBI.