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Armando Santoro

Researcher at Humanitas University

Publications -  961
Citations -  56759

Armando Santoro is an academic researcher from Humanitas University. The author has contributed to research in topics: Medicine & Cancer. The author has an hindex of 93, co-authored 847 publications receiving 48505 citations. Previous affiliations of Armando Santoro include Aix-Marseille University & University of Milan.

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Predictive Factors of Eribulin Activity in Metastatic Breast Cancer Patients.

TL;DR: Activity and safety profiles of eribulin in a real-world population of metastatic breast cancer patients were consistent with literature data and DFA could be a promising tool to discriminate responses to erIBulin among MBC patients.
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Phase II study of hypofractionated radiation therapy in elderly patients with newly diagnosed glioblastoma with poor prognosis.

TL;DR: HFRT has proven to be feasible and effective, with limited morbidity, for selected elderly and frail patients with newly diagnosed glioblastoma, and in selected patients, who need more aggressive treatment.
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Hypofractionation with simultaneous boost in breast cancer patients receiving adjuvant chemotherapy: A prospective evaluation of a case series and review of the literature.

TL;DR: Hypofractionated VMAT-SIB in patients who had undergone adjuvant systemic therapy was safe and well tolerated in terms of acute and early late settings and cosmesis and confirmed the results of other studies published on the association of hypofractionation and chemotherapy or concomitant boost.
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Retrospective Assessment of a Serum Proteomic Test in a Phase III Study Comparing Erlotinib plus Placebo with Erlotinib plus Tivantinib (MARQUEE) in Previously Treated Patients with Advanced Non-Small Cell Lung Cancer.

TL;DR: VeriStrat showed a prognostic role within Eastern Cooperative Oncology Group (ECOG) performance score (PS) categories and regardless of treatment arm and EGFR status, suggesting that Veri Strat could be used to identify EGFR mutation-positive patients who will have a poor response to EGFR tyrosine kinase inhibitors.