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Armando Santoro

Researcher at Humanitas University

Publications -  961
Citations -  56759

Armando Santoro is an academic researcher from Humanitas University. The author has contributed to research in topics: Medicine & Cancer. The author has an hindex of 93, co-authored 847 publications receiving 48505 citations. Previous affiliations of Armando Santoro include Aix-Marseille University & University of Milan.

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Clinical Review on the Management of Breast Cancer Visceral Crisis

TL;DR: In this article , the authors discuss the management of visceral crisis, advocating future treatment perspectives for this challenging condition, which is a life-threatening clinical condition requiring urgent treatment and accounts for 10-15% of new advanced breast cancer diagnoses, mainly hormone receptor positive/human epidermal growth factor 2 negative.

P27: humoral response rate post mrna covid19 vaccination (bnt162b2) is higly dependent to bone marrow plasmacytosis: long follow-up of symptomatic multiple myeloma patients

TL;DR: The myeloma treatment, including high-dose melphalan and autologous stem cell transplantation, have not been associated with SARS-CoV-2 infection and a significant fraction of MM patients does not developed any detectable anti-Spike IgG after two dose of COVID-19 mRNA vaccine.
Journal ArticleDOI

Selected memory T cells infused post haploidentical hematopoietic stem cell transplantation persist and hyper-expand.

TL;DR: In this paper , a longitudinal analysis of the lymphocyte compartment in 19 haplo-HSCT patients previously enrolled in a phase II prospective clinical trial (ClinicalTrials.gov Identifier: NCT04687982), in which they received post-transplant CD45RA-depleted donor lymphocyte infusions (DLI), was performed.
Posted ContentDOI

Clinical, pathological and molecular predictors of Fulvestrant long-term benefit in Hormone Receptor-positive / HER2 negative advanced breast cancer

TL;DR: The clinical findings are consistent with literature data, while the mutation and miRNA results provide some clues on the molecular mechanisms involved in fulvestrant activity and resistance to be confirmed in larger cohorts of breast cancer patients.