A
Arthur W. Wallace
Researcher at University of California, San Francisco
Publications - 88
Citations - 5431
Arthur W. Wallace is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Perioperative & Left Ventricular Aneurysm. The author has an hindex of 29, co-authored 88 publications receiving 5036 citations. Previous affiliations of Arthur W. Wallace include San Francisco VA Medical Center & University of California, Berkeley.
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Patent
L-arginine and phosphodiesterase (PDE) inhibitor synergism
TL;DR: In this paper, a combination of Larginine and type V phosphodiesterase inhibitors was used for the treatment of cardiac pathologies and/or for erectile dysfunction.
Journal ArticleDOI
Dor procedure for dyskinetic anteroapical myocardial infarction fails to improve contractility in the border zone.
Kay Sun,Kay Sun,Zhihong Zhang,Zhihong Zhang,Takamaro Suzuki,Takamaro Suzuki,Jonathan F. Wenk,Jonathan F. Wenk,Nielen Stander,Daniel R. Einstein,David Saloner,David Saloner,Arthur W. Wallace,Arthur W. Wallace,Julius M. Guccione,Julius M. Guccione,Mark B. Ratcliffe,Mark B. Ratcliffe +17 more
TL;DR: The Dor procedure decreases end-diastolic and end-systolic stress but fails to improve contractility in the infarct border zone, and future work should focus on measures that will enhance border zone function alone or in combination with surgical remodeling.
Journal ArticleDOI
Left Ventricular Myocardial Contractility Is Depressed in the Borderzone After Posterolateral Myocardial Infarction
Rafael Shimkunas,Zhihong Zhang,Jonathan F. Wenk,Mehrdad Soleimani,Michael Khazalpour,Gabriel Acevedo-Bolton,Guanying Wang,David Saloner,David Saloner,Rakesh Mishra,Rakesh Mishra,Arthur W. Wallace,Arthur W. Wallace,Liang Ge,Liang Ge,Anthony J. Baker,Anthony J. Baker,Julius M. Guccione,Julius M. Guccione,Mark B. Ratcliffe,Mark B. Ratcliffe +20 more
TL;DR: Contractility in the BZ is significantly depressed relative to the remote myocardium after posterolateral MI, and this reduction in contractility is due at least in part to a decrease in contractile protein function.
Journal ArticleDOI
Moderate mitral regurgitation accelerates left ventricular remodeling after posterolateral myocardial infarction.
Mehrdad Soleimani,Michael Khazalpour,Guangming Cheng,Zhihong Zhang,Gabriel Acevedo-Bolton,Gabriel Acevedo-Bolton,David Saloner,David Saloner,Rakesh Mishra,Rakesh Mishra,Arthur W. Wallace,Arthur W. Wallace,Julius M. Guccione,Julius M. Guccione,Liang Ge,Liang Ge,Mark B. Ratcliffe,Mark B. Ratcliffe +17 more
TL;DR: Moderate MR accelerates LV remodeling after posterolateral MI and further studies are needed to determine whether mitral valve repair is able to slow or reverse MI remodeling before and after postersolateral myocardial infarction.
Journal ArticleDOI
A novel method for quantifying in-vivo regional left ventricular myocardial contractility in the border zone of a myocardial infarction.
Lik Chuan Lee,Jonathan F. Wenk,Doron Klepach,Zhihong Zhang,David Saloner,Arthur W. Wallace,Liang Ge,Mark B. Ratcliffe,Julius M. Guccione +8 more
TL;DR: A mathematical model of a sheep LV having an anteroapical infarct with linearly-varying BZ contractility is developed and suggests that future mathematical LV models, particularly those used to analyze myocardial infarction treatment, should account for a smooth linear transition in contractility within the BZ.