Ashkan Farhadi

TL;DR: A review of the normal and abnormal functioning of the intestinal barrier, the diseases that can result from loss of barrier integrity, and some promising agents and strategies for restoring barrier normality and integrity are reviewed.

TL;DR: Probiotics could preserve normal barrier function in an animal model of ALD by preventing alcohol-induced oxidative stress and thus prevent the development of hyperpermeability and subsequent alcoholic steatohepatitis (ASH) in rats and provides a scientific rationale to test probiotics for treatment and/or prevention of alcoholic liver disease in man.

TL;DR: The hypothesis that gut leakiness resulting in endotoxemia is a key co-factor (trigger) for ASH is supported, as well as the hypothesis that alcohol-induced gut leakediness preceded steatohepatitis indicates they are not the consequence of ALD.

TL;DR: It is shown that there is a rather large number of theories on how cells can generate and detect electromagnetic fields and experimental evidence on electromagnetic cellular interactions in the modern scientific literature is continuously accumulating.

TL;DR: This work investigated one possible mechanism for the endotoxaemia – disruption of intestinal barrier integrity – and found it to be related to hepatic oxidative stress triggered by elevated levels of endotoxin.