B
Barbara A. Gower
Researcher at University of Alabama at Birmingham
Publications - 327
Citations - 13916
Barbara A. Gower is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Insulin & Insulin resistance. The author has an hindex of 59, co-authored 311 publications receiving 12520 citations. Previous affiliations of Barbara A. Gower include University of Alabama & University of Utah.
Papers
More filters
Journal ArticleDOI
Age-related decrease in resting energy expenditure in sedentary white women: effects of regional differences in lean and fat mass
TL;DR: T trunk lean mass (presumably primarily organ tissue) is relatively resistant to age-related changes in body composition, whereas muscle mass, especially leg muscle, tends to be lost.
Journal ArticleDOI
Higher Maternal Gestational Glucose Concentration Is Associated with Lower Offspring Insulin Sensitivity and Altered β-Cell Function
Nikki C. Bush,Paula C. Chandler-Laney,Dwight J. Rouse,Wesley M. Granger,Robert A. Oster,Barbara A. Gower +5 more
TL;DR: Maternal gestational glucose concentration was significantly associated with offspring insulin sensitivity and β-cell response independent of adiposity, suggesting that maternal glucose may program the fetus both at the pancreas and at the level of insulin target tissues such as skeletal muscle and liver.
Journal ArticleDOI
Do neighbourhoods matter? Neighbourhood disorder and long-term trends in serum cortisol levels
TL;DR: Children who are exposed to negative socioenvironmental climates over time are more likely to have altered serum cortisol levels, which may be an adaptive mechanism to cope with stress; however, disrupted cortisol levels may have negative effects on general physical and mental health.
Journal ArticleDOI
Adiponectin Is Lower Among African Americans and Is Independently Related to Insulin Sensitivity in Children and Adolescents
TL;DR: Positive correlated with S(i), inversely related to central fat, and positively related to peripheral fat were found, and higher acute insulin response to glucose explained lower adiponectin among African-American children.
Journal ArticleDOI
Relations of fatness and fitness to fasting insulin in black and white adolescents.
TL;DR: Interventions to prevent hyperinsulinemia in youths should be designed both to minimize fatness and maximize CVF.