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Barbara A. Gower

Researcher at University of Alabama at Birmingham

Publications -  327
Citations -  13916

Barbara A. Gower is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Insulin & Insulin resistance. The author has an hindex of 59, co-authored 311 publications receiving 12520 citations. Previous affiliations of Barbara A. Gower include University of Alabama & University of Utah.

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Body fat distribution in white and black women: different patterns of intraabdominal and subcutaneous abdominal adipose tissue utilization with weight loss

TL;DR: Despite comparable decreases in total and trunk fat, white women lost more IAAT and less SAAT than did black women and Waist circumference was not a suitable surrogate marker for tracking changes in the visceral fat compartment in black women.
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Racial differences in insulin secretion and sensitivity in prepubertal children: role of physical fitness and physical activity.

TL;DR: Overall physical activity and, especially, vigorous activity were associated with insulin secretion and sensitivity, however, neither physical activity nor VO2max explained the racial difference in insulin secretion (higher in African Americans) and sensitivity (lower in African American).
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Effect of dietary adherence with or without exercise on weight loss: a mechanistic approach to a global problem.

TL;DR: Dietary adherence is strongly associated with rates of weight loss and adversely affected by the severity of caloric restriction, and weight loss programs should consider moderate caloric restriction relative to estimates of energy requirements, rather than generic low-calorie diets.
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Threshold for Effects of Vitamin D Deficiency on Glucose Metabolism in Obese Female African-American Adolescents

TL;DR: Vitamin D deficiency is highly prevalent in obese, AA female adolescents and may promote insulin resistance and the data suggest that a 25(OH)D concentration of 15 ng/ml or less may be the threshold by which vitamin D deficiency confers negative effects on insulin sensitivity.