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Barbara De Conno

Researcher at University of Naples Federico II

Publications -  9
Citations -  136

Barbara De Conno is an academic researcher from University of Naples Federico II. The author has contributed to research in topics: Proinflammatory cytokine & Receptor. The author has an hindex of 4, co-authored 7 publications receiving 50 citations.

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Diet and functional dyspepsia: Clinical correlates and therapeutic perspectives

TL;DR: An overview of the evidence-based nutritional approach that should be pursued in functional dyspepsia patients, providing clinicians with a valuable tool in standardizing nutritional advises and discouraging patients from engaging into indiscriminate food exclusions.
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S100B Protein Stimulates Proliferation and Angiogenic Mediators Release through RAGE/pAkt/mTOR Pathway in Human Colon Adenocarcinoma Caco-2 Cells.

TL;DR: Results showed that S100B markedly increases proliferation and invasiveness of Caco-2 cells, through the release of pro-angiogenic VEGF and NO paralleled to a significant decrease of wtp53 expression mediated by RAGE-p38 mitogen-activated protein kinase (MAPK)/pAkt-mTOR and hypoxia-inducible factor 1-alpha (HIF1α) pathways.
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Nutritionist and obesity: brief overview on efficacy, safety, and drug interactions of the main weight-loss dietary supplements.

TL;DR: The aim of this review was to provide a synthetic, evidence-based report on efficacy and safety of the most commonly used ingredients in dietary supplements marketed for weight loss, particularly focusing on their possible drug interactions.
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Ultramicronized Palmitoylethanolamide Inhibits NLRP3 Inflammasome Expression and Pro-Inflammatory Response Activated by SARS-CoV-2 Spike Protein in Cultured Murine Alveolar Macrophages.

TL;DR: In this article, the anti-inflammatory and immunomodulatory activity of ultramicronized palmitoylethanolamide ( um)-PEA, both alone and in the presence of specific peroxisome proliferator-activated receptor alpha (PPAR-α) antagonist MK886, in primary cultures of murine alveolar macrophages exposed to SARS-CoV-2 spike glycoprotein (SP).