B
Bert Bammens
Researcher at Katholieke Universiteit Leuven
Publications - 142
Citations - 6766
Bert Bammens is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Transplantation & Kidney disease. The author has an hindex of 44, co-authored 129 publications receiving 5733 citations. Previous affiliations of Bert Bammens include Catholic University of Leuven.
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Oxidative stress in chronic kidney disease.
Kristien Daenen,Kristien Daenen,Asmin Andries,Djalila Mekahli,Ann Van Schepdael,François Jouret,Bert Bammens +6 more
TL;DR: The potential role of various antioxidants and pharmacological agents, which may represent potential therapeutic targets to reduce OS in both pediatric and adult CKD patients, are discussed.
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Uremic toxins originating from colonic microbial metabolism
TL;DR: There is evidence that besides reduced renal clearance, increased colonic generation and absorption explain the high levels of bacterial URMs in CKD.
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Free serum concentrations of the protein-bound retention solute p-cresol predict mortality in hemodialysis patients
TL;DR: The data suggest that free serum levels of p-cresol, a representative of the protein-bound uremic retention solutes, are associated with mortality in HD patients, and may encourage nephrologists to widen their field of interest beyond the scope of small water-soluble ureming solutes and middle molecules.
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Free p-cresol is associated with cardiovascular disease in hemodialysis patients
TL;DR: It is shown that free p-cresol is a novel cardiovascular risk factor in non-diabetic hemodialysis patients and in diabetic patients, however, a significant relationship between p-Cresol and cardiovascular events could not be demonstrated despite their having significantly higher p- cresol levels.
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Natural history of parathyroid function and calcium metabolism after kidney transplantation: a single-centre study
TL;DR: Kidney transplant recipients with a high iPTH and calcium x phosphate product at the time of transplantation are at risk for persistent hyperparathyroidism, especially when renal function is suboptimal.