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Bhavani S. Sahu

Researcher at Indian Institute of Technology Madras

Publications -  20
Citations -  335

Bhavani S. Sahu is an academic researcher from Indian Institute of Technology Madras. The author has contributed to research in topics: Receptor & Chromogranin A. The author has an hindex of 11, co-authored 16 publications receiving 273 citations. Previous affiliations of Bhavani S. Sahu include University of Cambridge & University of California, San Diego.

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Enhancement in the efficiency of polymerase chain reaction by TiO2 nanoparticles: crucial role of enhanced thermal conductivity.

TL;DR: TiO(2) nanoparticle-assisted PCR may be useful for profound reduction of the overall PCR reaction period and for enhanced amplification of DNA amplicons from a variety of samples, including GC-rich templates that are often observed to yield unsatisfactory results.
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Coordinated transcriptional regulation of Hspa1a gene by multiple transcription factors: crucial roles for HSF-1, NF-Y, NF-κB, and CREB.

TL;DR: Previously unknown roles for NF-κB and CREB to regulate Hspa1a expression and a coordinated action by several transcription factors for HspA1a transactivation under heat-shock/ischemia-like conditions are discovered and thereby provided new insights into the mechanism of Hsp a1a regulation.
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Transcriptional regulation of the novel monoamine oxidase renalase: Crucial roles of transcription factors Sp1, STAT3, and ZBP89

TL;DR: Interestingly, renalase promoter activity was augmented by nicotine and catecholamines; while Sp1 and STAT3 synergistically activated the nicotine-induced effect, Sp1 appeared to enhance epinephrine-evoked renalase transcription.
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Chromogranin A: A novel susceptibility gene for essential hypertension

TL;DR: Accumulated evidence establishes CHGA as a novel susceptibility gene for EH, which acts as a prohormone giving rise to bioactive peptides such as vasostatin-I and catestatin that exhibit several cardiovascular regulatory functions.
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The neuropeptide TLQP-21 opposes obesity via C3aR1-mediated enhancement of adrenergic-induced lipolysis.

TL;DR: TLQP-21 does not possess lipolytic properties per se, but it enhances β-AR activation-induced lipolysis by a mechanism requiring Ca2+ mobilization and ERK activation of Hormone Sensitive Lipase (HSL).