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Bin Xiang

Researcher at South China Agricultural University

Publications -  40
Citations -  464

Bin Xiang is an academic researcher from South China Agricultural University. The author has contributed to research in topics: Virus & Newcastle disease. The author has an hindex of 11, co-authored 31 publications receiving 300 citations. Previous affiliations of Bin Xiang include Chinese Ministry of Agriculture.

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Phylogenetic and Pathotypic Characterization of Newcastle Disease Viruses Circulating in South China and Transmission in Different Birds

TL;DR: It is indicated that genetically diverse viruses circulate in LBMs in South China's Guangdong Province and that NDV from different birds have different tissue tropisms and host ranges when transmitted in different birds.
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Newcastle disease virus infection in chicken embryonic fibroblasts but not duck embryonic fibroblasts is associated with elevated host innate immune response

TL;DR: Results show distinct expression patterns of cytokines, Toll-like receptors and IFNs associated with inflammatory immune responses to NDV between species and by virulence.
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Pathogenicity and transmissibility of three avian influenza A (H5N6) viruses isolated from wild birds

TL;DR: The results raise the possibility that wild birds disseminate these H5N6 HPAIVs to poultry along their flyways and thus pose a great threat to the poultry industry and, thus, public health.
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Host Innate Immune Responses of Ducks Infected with Newcastle Disease Viruses of Different Pathogenicities.

TL;DR: Comparisons of viral distribution and mRNA expression of immune-related genes in ducks following infection with two NDV strains highlight important differences in host innate immune responses, courses of infection, and pathogenesis following NDV infection.
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Newcastle disease virus-induced autophagy mediates antiapoptotic signaling responses in vitro and in vivo.

TL;DR: It is demonstrated that apoptosis inhibition enhances autophagy and promoted cell survival and NDV replication and cell viability by inhibiting apoptosis.