Bing Ching Ho

TL;DR: The results suggest that upregulation of miR-141 upon enterovirus infection can facilitate viral propagation by expediting the translational switch, and identify a transcription factor, EGR1, which is partly responsible for mi R-141 induction in response to enterov virus infection.

TL;DR: YAP1 mutation was shown to increase the colony formation ability and invasion potential of lung cancer cells and implicated YAP1 R331W as an allele predisposed for lung adenocarcinoma with high familial penetrance.

TL;DR: The hES‐T3 cells with normal female karyotype cultured on either mouse embryonic fibroblasts in hES medium or feeder‐free Matrigel in MEF‐conditioned medium were found to express very similar profiles of mRNAs and microRNAs, indicating that the unlimited self‐renewal and pluripotency of hES cells can be maintained by continuing culture on these two conditions.

TL;DR: It is shown that EV71‐induced cell apoptosis is partly governed by altered miRNAs, and the transfection of antagomiR‐146a and miR‐370 contributed to attenuating EV71 infection‐induced apoptosis.

TL;DR: An online system, ChemiRs, which contains miRNAs, diseases, Medical Subject Heading (MeSH) terms, chemicals, genes, pathways and PubMed IDs, and is freely available for public use at http://omics.ntnu.edu.tw/ChemiRs.