B
Bolek Srebro
Researcher at University of Bergen
Publications - 16
Citations - 1475
Bolek Srebro is an academic researcher from University of Bergen. The author has contributed to research in topics: Long-term potentiation & Excitatory postsynaptic potential. The author has an hindex of 15, co-authored 16 publications receiving 1455 citations.
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Journal ArticleDOI
Changes in acetylcholinesterase and distribution of degenerating fibres in the hippocampal region after septal lesions in the rat.
Sven Ivar Mellgren,Bolek Srebro +1 more
TL;DR: The distribution of acetylcholinesterase and loss of enzyme in different layers and areas after medial septal lesions were correlated with the distribution of degenerating axons and terminals in Fink-Heimer preparations of the same material.
Journal ArticleDOI
Acute Intrahippocampal Infusion of BDNF Induces Lasting Potentiation of Synaptic Transmission in the Rat Dentate Gyrus
TL;DR: It is concluded that exogenous BDNF induces a lasting potentiation of synaptic efficacy in the dentate gyrus of anesthetized adult rats.
Journal ArticleDOI
Behavioral effects of selective midbrain raphe lesions in the rat.
Bolek Srebro,Stanley A. Lorens +1 more
TL;DR: It is suggested, furthermore, that the behavioral effects of midbrain raphe lesions are not due primarily to their associated reduction in forebrain 5-HT, as well as their apparent similarity to the effects of other mesencephalic and limbic lesions.
Journal ArticleDOI
Synaptic plasticity in the hippocampus is modulated by behavioral state.
Clive R. Bramham,Bolek Srebro +1 more
TL;DR: The results suggest that synaptic plasticity is dynamically modulated during the sleep-wakefulness cycle, and the efficacy of low-frequency neurotransmission varied with behavioral state.
Journal ArticleDOI
Activity, avoidance learning and regional 5-hydroxytryptamine following intra-brain stem 5,7-dihydroxytryptamine and electrolytic midbrain raphe lesions in the rat.
TL;DR: The hyperactivity and impaired one-way avoidance performance observed after electrolytic midbrain raphe lesions are not related simply to reductions in regional forebrain 5-HT and may well be due to damage of non-serotonergic neural systems.