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Borna Relja
Researcher at Otto-von-Guericke University Magdeburg
Publications - 142
Citations - 3612
Borna Relja is an academic researcher from Otto-von-Guericke University Magdeburg. The author has contributed to research in topics: Inflammation & Lung injury. The author has an hindex of 25, co-authored 128 publications receiving 2387 citations. Previous affiliations of Borna Relja include Goethe University Frankfurt & Mie University.
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Journal ArticleDOI
Cytokines in Inflammatory Disease.
TL;DR: This review aims to briefly discuss a short list of a broad variety of inflammatory cytokines, focusing on the correlations and role of these inflammatory mediators in the genesis of inflammatory impacts.
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CXCR4 Chemokine Receptor Mediates Prostate Tumor Cell Adhesion through α5 and β3 Integrins
Tobias Engl,Borna Relja,Dana Marian,Christa Blumenberg,Iris Müller,Wolf-Dietrich Beecken,Jon Jones,Eva M. Ringel,Jürgen Bereiter-Hahn,Dietger Jonas,Roman A. Blaheta +10 more
TL;DR: Data show that chemoattractive mechanisms are involved in adhesion processes of prostate cancer cells, and that binding of CXCL12 to its receptor leads to enhanced expression of α 5 and β 3 .
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Establishment and characterization of the Masquelet induced membrane technique in a rat femur critical‐sized defect model
Dirk Henrich,Caroline Seebach,Christoph Nau,S. Basan,Borna Relja,Kerstin Wilhelm,Alexander Schaible,Johannes Frank,John H. Barker,Ingo Marzi +9 more
TL;DR: It was concluded that cellular composition and growth factor content in induced membranes depends on the location where the membrane is induced and differs from periosteum.
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Damage-associated molecular patterns in trauma.
Borna Relja,Walter Land +1 more
TL;DR: This review focuses on the recent literature dealing with the pathophysiological importance of DAMPs after traumatic injury, and so-called model of suppressing/inhibiting inducible DAMPs (SAMPs) will be very briefly introduced.
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Simvastatin inhibits cell growth and induces apoptosis and G0/G1 cell cycle arrest in hepatic cancer cells
TL;DR: The results suggest that simvastatin induces apoptosis in tumor cells and its anti-proliferative activity was accompanied by inhibition of cyclin-dependent kinases and cyclins, whereas CDK inhibitors p19 and p27 were enhanced.