Brenda J. Tripathi

TL;DR: The present findings support the concept of the intraocular derivation of this cytokine and hypothesize that the increased level of TGF-β2 in the aqueous humor of POAG patients causes a decrease in the cellularity of the trabecular meshwork and promotes the buildup of excessive amounts of ECM materials that are characteristic of this disease and contribute to the increased resistance to aQueous outflow.

TL;DR: Bony orbit and paranasal sinuses Ocular appendages Orbital and cerebral vessels Extraocular muscles and ocular movements Innervation and nerves of the orbit The eyeball and its dimensions Cornea and sclera Anterior chamber and drainage angle The iris Posterior chamber and ciliary body Choroid and uveal vessels Lens and zonules The vitreous The retina Visual pathway Autonomic aminergic, peptidergic and nitrergic innervation of the human eye as mentioned in this paper.

TL;DR: Several hypotheses regarding the pathogenesis of Fuchs' dystrophy are discussed, including the possible influences of aberrant embryogenesis, hormones, and injury on the development of the disease.

TL;DR: The authors' findings show that patients with NVG had a significantly increased level of VEGF in the aqueous humor and implicate V EGF as an important factor in the pathogenesis of intraocular neovascularization in these patients.

TL;DR: The proposed mechanism of corticosteroid-induced glaucoma includes morphological and functional changes in the trabecular meshwork system and is similar to the pathogenesis of POAG.