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Brian Walitt

Researcher at National Institutes of Health

Publications -  95
Citations -  4409

Brian Walitt is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Fibromyalgia & Women's Health Initiative. The author has an hindex of 31, co-authored 84 publications receiving 3247 citations. Previous affiliations of Brian Walitt include MedStar Washington Hospital Center & George Washington University.

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The Prevalence and Characteristics of Fibromyalgia in the 2012 National Health Interview Survey

TL;DR: Examination of the surrogate polysymptomatic distress scale (PSD) of the 2010 ACR criteria found fibromyalgia symptoms extending through the full length of the scale with physiological as well as mental stressors suggests PSD may be a universal response variable rather than one restricted to Fibromyalgia.
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Fibromyalgia diagnosis and biased assessment: Sex, prevalence and bias.

TL;DR: The perception of fibromyalgia as almost exclusively a women’s disorder is not supported by data in unbiased studies, and the use of 2016 fibromy nostalgia criteria for clinical diagnosis and epidemiology is recommended because of its updated scoring and generalized pain requirement.
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Chemobrain: A critical review and causal hypothesis of link between cytokines and epigenetic reprogramming associated with chemotherapy

TL;DR: It is hypothesized that the administration of chemotherapy agents initiates a cascade of biological changes, with short-lived alterations in the cytokine milieu inducing persistent epigenetic alterations that lead to changes in gene expression, alterations in metabolic activity and neuronal transmission responsible for generating the subjective experience of cognition.
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The development of fibromyalgia--I: examination of rates and predictors in patients with rheumatoid arthritis (RA).

TL;DR: Study results indicate that multiple, inter‐correlated factors that include social disadvantage, psychological distress, comorbidity, RA severity, and fibromyalgia variables predict future development of fibromyalgia, but there is little evidence of the effect of underlying causes.