Z
Zhang-Jin Zhang
Researcher at University of Hong Kong
Publications - 219
Citations - 6213
Zhang-Jin Zhang is an academic researcher from University of Hong Kong. The author has contributed to research in topics: Randomized controlled trial & Medicine. The author has an hindex of 36, co-authored 176 publications receiving 4777 citations. Previous affiliations of Zhang-Jin Zhang include National Institutes of Health & Vanderbilt University.
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The Role of Oxidative Stress and Antioxidants in Liver Diseases
TL;DR: Animal studies have revealed the promising in vivo therapeutic effect of antioxidants on liver diseases, and various factors that cause oxidative stress in liver and effects of antioxidants in the prevention and treatment of liver diseases were summarized, questioned, and discussed.
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Therapeutic effects of herbal extracts and constituents in animal models of psychiatric disorders.
TL;DR: Herbal remedies that have demonstrable psychotherapeutic activities have provided a potential to psychiatric pharmaceuticals and deserve increased attention in future studies.
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Neural acupuncture unit: a new concept for interpreting effects and mechanisms of acupuncture.
TL;DR: The introduction of the conception of NAU provides a new theoretical approach to interpreting effects and mechanisms of acupuncture in modern biomedical knowledge framework.
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The effectiveness and safety of acupuncture therapy in depressive disorders: Systematic review and meta-analysis
TL;DR: The efficacy of acupuncture as monotherapy was comparable to antidepressants alone in improving clinical response and alleviating symptom severity of MDD, but not different from sham acupuncture.
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Chemobrain: A critical review and causal hypothesis of link between cytokines and epigenetic reprogramming associated with chemotherapy
TL;DR: It is hypothesized that the administration of chemotherapy agents initiates a cascade of biological changes, with short-lived alterations in the cytokine milieu inducing persistent epigenetic alterations that lead to changes in gene expression, alterations in metabolic activity and neuronal transmission responsible for generating the subjective experience of cognition.