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Bruna M Mazetto

Researcher at State University of Campinas

Publications -  34
Citations -  290

Bruna M Mazetto is an academic researcher from State University of Campinas. The author has contributed to research in topics: Antiphospholipid syndrome & Thrombosis. The author has an hindex of 8, co-authored 28 publications receiving 210 citations.

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Folate, Vitamin B12 and Homocysteine status in the post-folic acid fortification era in different subgroups of the Brazilian population attended to at a public health care center

TL;DR: The results suggest that folate deficiency is practically nonexistent in the post-folic acid fortification era in the subgroups evaluated, however, screening for vitamin B12 deficiency may be particularly relevant in the elderly.
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Reduced thrombin formation and excessive fibrinolysis are associated with bleeding complications in patients with dengue fever: a case–control study comparing dengue fever patients with and without bleeding manifestations

TL;DR: DF can manifest with spontaneous bleedings, which are associated with specific coagulation and fibrinolysis profiles that are not significantly present in DF without this complication, and thrombocytopenia, excessive fibrinelysis and reduced thrombin formation may contribute to the bleeding manifestations in DF.
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Recurrent thrombosis in antiphospholipid syndrome may be associated with cardiovascular risk factors and inflammatory response.

TL;DR: The results suggest that arterial hypertension and monocyte counts may be independent factors for thrombosis recurrence in APS, and may support the evaluation of therapeutic measures to a rigid control of blood pressures and modulation of inflammatory response in APs, as additional prophylaxis against the recurrence of vascular events.
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Increased ADAMTS13 activity in patients with venous thromboembolism

TL;DR: Increased levels of inflammatory markers and VWF may suggest that there is a persistence of inflammatory activity in patients even at long periods after the VTE episode, and it may be postulated that increased levels of ADAMTS13 could represent a compensatory mechanism against persistently increased Levels of VWF.
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Long-term increased factor VIII levels are associated to interleukin-6 levels but not to post-thrombotic syndrome in patients with deep venous thrombosis

TL;DR: It is demonstrated that DVT is associated with increased levels of inflammatory and coagulation markers, including FVIII, even a long time after the acute episode, and PTS seems to be related to inflammatory cytokine IL-8, a proinflammatory and proangiogenic chemokine, but not to FV III levels.