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Joyce Annichino-Bizzachi

Researcher at State University of Campinas

Publications -  8
Citations -  145

Joyce Annichino-Bizzachi is an academic researcher from State University of Campinas. The author has contributed to research in topics: Antiphospholipid syndrome & Risk factor. The author has an hindex of 4, co-authored 8 publications receiving 118 citations.

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Journal ArticleDOI

Cerebral venous thrombosis: influence of risk factors and imaging findings on prognosis.

TL;DR: MRI is superior to conventional CT for diagnosing CVT and patients with parenchymal lesions, thrombophilia and antiphospholipid syndrome had greater risk to be left with neurological sequelae.
Journal ArticleDOI

Recurrent thrombosis in antiphospholipid syndrome may be associated with cardiovascular risk factors and inflammatory response.

TL;DR: The results suggest that arterial hypertension and monocyte counts may be independent factors for thrombosis recurrence in APS, and may support the evaluation of therapeutic measures to a rigid control of blood pressures and modulation of inflammatory response in APs, as additional prophylaxis against the recurrence of vascular events.
Book ChapterDOI

Use of Platelet-Rich Plasma (PRP) in Treating Chronic Wounds

TL;DR: Platelet-leukocyte gel (L-PRP), besides releasing the growth factors that start tissue regeneration, can also strengthen the antimicrobial activity, which shows its potential as an infection prevention and treatment agent.
Journal ArticleDOI

Circulating levels of tissue factor and the risk of thrombosis associated with antiphospholipid syndrome.

TL;DR: Circulating TF is associated with thrombotic complications related to APS, but not with the risk of unprovoked VTE, as compared with controls.
Journal ArticleDOI

Acquired thrombotic thrombocytopenic purpura due to antibody-mediated ADAMTS13 deficiency precipitated by a localized Castleman's disease: a case report.

TL;DR: A case of refractory TTP precipitated by a newly diagnosed localized Castleman’s disease (CD) and the antibody to ADAMTS13 developed along with the systemic manifestation of CD and promptly disappeared after the resection of the tumor.