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Camille Piguet

Researcher at University of Geneva

Publications -  56
Citations -  886

Camille Piguet is an academic researcher from University of Geneva. The author has contributed to research in topics: Bipolar disorder & Mood. The author has an hindex of 13, co-authored 46 publications receiving 635 citations. Previous affiliations of Camille Piguet include Université Paris-Saclay & University of Strasbourg.

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Methylation of serotonin receptor 3a in adhd, borderline personality, and bipolar disorders: link with severity of the disorders and childhood maltreatment.

TL;DR: The impact of childhood maltreatment on the methylation status of the 5‐HT3AR and its association with clinical severity outcomes in relation with a functional genetic polymorphism is studied.
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Childhood maltreatment and methylation of the glucocorticoid receptor gene NR3C1 in bipolar disorder

TL;DR: Early-life adversities have a sustained effect on the HPA axis through epigenetic processes and this effect may be measured in peripheral blood.
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Resting-state functional connectivity of emotion regulation networks in euthymic and non-euthymic bipolar disorder patients.

TL;DR: The results suggest that sgACC-amygdala coupling is critically affected during mood episodes, and that FC of sg ACC play a pivotal role in mood normalization through its interactions with the VLPFC and PCC.
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What makes your brain suggestible? Hypnotizability is associated with differential brain activity during attention outside hypnosis.

TL;DR: Outside hypnosis, low hypnotizable subjects recruited more parietal cortex and anterior cingulate regions during selective attention conditions suggesting a better detection and implementation of conflict, and points to specific neural substrates in right prefrontal cortex.
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Modulation of brain response to emotional conflict as a function of current mood in bipolar disorder: preliminary findings from a follow-up state-based fMRI study.

TL;DR: The results indicate that the ability of BD patients to recruit control networks when processing affective conflict, and the abnormal suppression of activity in distinct components of the default mode network, may depend on their current clinical state and attentional demand.