Caterina Morabito

TL;DR: The data support a possible link between exposure to ELF-EMFs and modification of the cellular redox state, which could, in turn, increase the level of intracellular Ca(2+) and thus modulate the metabolic activity of C2C12 cells.

TL;DR: The study indicates that AD-related mutations may promote enhanced ROS generation, oxidative-dependent oligomerization, and loss of function of Kv2.1 channels, which may set a deleterious vicious circle that eventually helps to promote excitotoxic damage found in the AD brain.

TL;DR: The hypothesis that ROS and Ca2+ could be the cellular “primum movens” of the ELF-EMF induced effects on biological systems is supported.

TL;DR: Proliferation assays and cytofluorimetric analyses indicated a significant anti‐proliferative effect of purines, and a concentration‐dependent accumulation of cells in S‐phase, starting after 24 h of purine exposure and extending for up to 6 days.

TL;DR: It is shown that the sarcolemmal conduction system and some aspects of Ca(2+) transport are negatively influenced in chronic fatigue syndrome, and both deregulation of pump activities and alteration in the opening status of ryanodine channels may result from increased membrane fluidity involving sarcoplasmic reticulum membranes.