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Silvia Belia
Researcher at University of Perugia
Publications - 38
Citations - 1467
Silvia Belia is an academic researcher from University of Perugia. The author has contributed to research in topics: Oxidative stress & Skeletal muscle. The author has an hindex of 18, co-authored 38 publications receiving 1365 citations. Previous affiliations of Silvia Belia include University of Chieti-Pescara.
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Journal ArticleDOI
Skeletal muscle is a primary target of SOD1G93A-mediated toxicity.
Gabriella Dobrowolny,Michela Aucello,Emanuele Rizzuto,Sara Beccafico,Cristina Mammucari,Simona Bonconpagni,Silvia Belia,Francesca Wannenes,Carmine Nicoletti,Zaccaria Del Prete,Nadia Rosenthal,Mario Molinaro,Feliciano Protasi,Giorgio Fanò,Marco Sandri,Antonio Musarò,Antonio Musarò +16 more
TL;DR: It is demonstrated that skeletal muscle is a primary target of SOD1(G93A) -mediated toxicity and the molecular mechanism whereby oxidative stress triggers muscle atrophy is disclosed.
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The S-100: a protein family in search of a function.
TL;DR: "In vitro" and "in vivo" experiments showed that lower (nM) concentrations of extracellular S-100 beta act on glial and neuronal cells as a growth-differentiating factor and higher concentrations of the protein induce apoptosis of some cells such as the sympathetic-like PC12 line.
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Age and sex influence on oxidative damage and functional status in human skeletal muscle.
Giorgio Fanò,Patrizia Mecocci,Jacopo Vecchiet,Silvia Belia,Stefania Fulle,M. Cristina Polidori,Giorgio Felzani,Umberto Senin,Leonardo Vecchiet,M. Flint Beal +9 more
TL;DR: It is concluded that oxidative stress play an important role in muscle aging and that oxidative damage is much more evident in elderly males, suggesting a gender difference maybe related to hormonal factors.
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Dexamethasone in Glioblastoma Multiforme Therapy: Mechanisms and Controversies.
Marta Cenciarini,Mario Valentino,Silvia Belia,Luigi Sforna,Paolo Rosa,Simona Ronchetti,Maria Cristina D'Adamo,Mauro Pessia,Mauro Pessia +8 more
TL;DR: The main clinical features and genetic and epigenetic abnormalities underlying GBM, as well as current notions and concerns related to DEX effects on cerebral oedema, cancer cell proliferation and migration and clinical outcome are reviewed.
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Salicylates Inhibit Adhesion and Transmigration of T Lymphocytes by Preventing Integrin Activation Induced by Contact With Endothelial Cells
Roberto Gerli,Cristina Paolucci,Paolo Gresele,Onelia Bistoni,Stefano Fiorucci,C Muscat,Silvia Belia,Alberto Bertotto,Vincenzo Costantini +8 more
TL;DR: The data suggest that the antiinflammatory activity of salicylates may be due, at least in part, to an interference with the integrin-mediated binding of resting T lymphocytes to activated endothelium with consequent reduction of a specific T-cell recruitment into inflammatory sites.