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Catherine McDermott

Researcher at Bond University

Publications -  63
Citations -  5581

Catherine McDermott is an academic researcher from Bond University. The author has contributed to research in topics: Pyocyanin & Acetylcholine. The author has an hindex of 15, co-authored 60 publications receiving 4441 citations. Previous affiliations of Catherine McDermott include University College Cork.

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Journal ArticleDOI

Effect of short-term androgen deficiency on bladder contractility and urothelial mediator release.

TL;DR: Changes in mediator release may be an early effect of the decline in testosterone and could affect sensory pathways in the longer term, contributing to the urinary symptoms and bladder dysfunction seen in androgen-deficient men.
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The anxiolytic sertraline reduces the impact of psychological stress on bladder function in mice.

TL;DR: In this article, Sertraline was used to treat the bladder dysfunction caused by water avoidance stress in mice, and a blood sample was taken to measure plasma corticosterone levels and bladders were removed, catheterised and intravesical pressure responses recorded during distension and in response to pharmacological agents.
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The COX-2 inhibitor NS398 selectively sensitizes hypoxic HeLa cells to ionising radiation by mechanisms both dependent and independent of COX-2.

TL;DR: The aim of this study was to determine the impact of COX-2 selective inhibitors on both normoxic and hypoxic radiosensitivity in vitro and the mechanisms underlying this and to investigate their contribution to radioresistance.
Proceedings ArticleDOI

The evaluation of educational benefits of online learning tools on student performance in a major assessment item across two higher education institutions in australia

TL;DR: In this paper, a retrospective qualitative and quantitative study was conducted to evaluate the impact of adding e-tools, as supplements to the pharmacology curriculums in 2012, on student learning across two Australian universities; Griffith and Bond.
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Elevated release of inflammatory but not sensory mediators from the urothelium is maintained following epirubicin treatment.

TL;DR: Epirubicin treatment induces several transient changes in urothelial function, however, the increased secretion of inflammatory cytokines (IL-6 and IL-8) is sustained and these mediators may be involved in the pathophysiology of bladder toxicity following intravesical epirubic in treatment.