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Charles M. Greenspon

Researcher at University of Chicago

Publications -  19
Citations -  139

Charles M. Greenspon is an academic researcher from University of Chicago. The author has contributed to research in topics: Somatosensory system & Biology. The author has an hindex of 6, co-authored 11 publications receiving 55 citations. Previous affiliations of Charles M. Greenspon include University of Nottingham.

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Effect of scanning speed on texture-elicited vibrations

TL;DR: The objective of the present study is to characterize the effect of changes in scanning speed on texture-elicited vibrations to better understand how the exploratory movements shape the neuronal representation of texture.
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Chronic Use of a Sensitized Bionic Hand Does Not Remap the Sense of Touch.

TL;DR: In this paper, after long-term use of a neuromusculoskeletal prosthesis that featured a mismatch between the sensor location and the resulting tactile experience, the perceived location of the touch did not change.
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Age dependent plasticity in endocannabinoid modulation of pain processing through postnatal development

TL;DR: Study of the changing role of the ECs in the brainstem nuclei essential for the control of nociception from birth to adulthood in both rats and humans reveals that significant changes on ECs that to this point have been unknown are revealed and shed new light into the complex neurochemical changes that permit normal, mature responses to pain.
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Developmental alterations in noxious-evoked EEG activity recorded from rat primary somatosensory cortex

TL;DR: Data show that there are significant alterations in the processing of nociceptive inputs within the maturing cortex and that cortical theta activity is involved only in the adult cortical response to noxious stimulation.
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Manganese-enhanced magnetic resonance imaging depicts brain activity in models of acute and chronic pain: a new window to study experimental spontaneous pain?

TL;DR: The data suggest that on‐going pain related signal changes identified using MEMRI offers a new window to study the neural underpinnings of spontaneous pain in rats, and suggests reduced regional activation as a result of the intensity and duration of pain experienced during the 7 days of MnCl2 exposure.