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Chris D. Frith

Researcher at University College London

Publications -  526
Citations -  138274

Chris D. Frith is an academic researcher from University College London. The author has contributed to research in topics: Prefrontal cortex & Cognition. The author has an hindex of 173, co-authored 524 publications receiving 130472 citations. Previous affiliations of Chris D. Frith include Hammersmith Hospital & National Research Foundation of South Africa.

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Neurobiology: feeling right about doing right.

TL;DR: Results of work with brain-damaged patients constitute one line of evidence that the emotional component of judgements is not to be dismissed.
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Thematic Reasoning and Theory of Mind. Accounting for Social Inference Difficulties in Schizophrenia

TL;DR: Evidence in the schizophrenia data was provided to support the hypothesis of a relationship between theory of mind and social conditional reasoning and provided further support for the idea that in patients with schizophrenia at least, judgements about the mental states of others are achieved using analogical reasoning.
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Effect of the 5-HT1A partial agonist buspirone on regional cerebral blood flow in man

TL;DR: It is concluded that buspirone-induced alterations in regional cerebral blood flow are better understood, not in relation to the known distribution of monoamine neurotransmitter systems (particularly ascending 5-HT projections), but rather in relationto putative neuronal circuits possibly many synapses “downstream” of buspir one's pharmacological site of action.
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An fMRI study of joint action-varying levels of cooperation correlates with activity in control networks

TL;DR: The results indicate that, in the current experimental paradigm involving joint control of a visually presented object with joystick movements, the level of cooperation affected brain networks involved in action control, but not mentalizing.
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A biological marker for dyslexia.

Chris D. Frith, +1 more
- 04 Jul 1996 - 
TL;DR: Brain imaging reveals that, in dyslexics, the visual-motion area of the brain fails to activate, which may be a marker for a deviation in brain function which remained largely invisible until complex writing systems evolved.