C
Christian Sell
Researcher at Drexel University
Publications - 99
Citations - 15242
Christian Sell is an academic researcher from Drexel University. The author has contributed to research in topics: Senescence & Growth factor. The author has an hindex of 36, co-authored 96 publications receiving 13442 citations. Previous affiliations of Christian Sell include Lankenau Institute for Medical Research & Thomas Jefferson University.
Papers
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Journal ArticleDOI
DNA damage-induced degradation of Sp1 promotes cellular senescence
TL;DR: In this paper, the authors investigated the potential contribution of Sp1's role as a double strand break repair factor in mediating cellular senescence, and showed that Sp1 expression is decreased with a concomitant increase in senescent markers in response to DNA damage.
Journal Article
The role of the promoter in the expression of the PCNA gene.
Renee-Helene Charollais,Hansjuerg Alder,Andres Ferber,Janice Koniecki,Christian Sell,Renato Baserga +5 more
TL;DR: It is concluded that the failure of expression of the PCNA gene in G1-arrested TK-ts13 cells occurs at the transcriptional level.
Patent
Novel compositions and methods for treating or preventing dermal disorders
TL;DR: In this article, the authors present compositions and methods for treating or preventing certain dermal disorders including dermal atrophy, pseudoscars, actinic keratosis, seborrheic or actinic Keratoses, lentigines, focal areas of dermal thickening, and coarse wrinkles.
Journal ArticleDOI
Mitochondrial Haplogroup Influences Motor Function in Long-Term HIV-1-Infected Individuals.
Ashley Azar,Kathryn N. Devlin,Joshua Chang Mell,Tania Giovannetti,Vanessa Pirrone,Michael R. Nonnemacher,Shendra Passic,Katherine Kercher,Jean W. Williams,J.M. Jacobson,Brian Wigdahl,Brian Wigdahl,Will Dampier,David J. Libon,Christian Sell +14 more
TL;DR: Evidence that mitochondrial haplogroup is related to neuropsychological test performance among patients in chronic disease settings such as HIV-1 infection is provided, suggesting that this haplogroups provides a protective advantage when faced with the combined stress of HIV- 1 infection and long-term antiretroviral therapies.