C
Christopher C. Oberley
Researcher at University of Wisconsin-Madison
Publications - 4
Citations - 63
Christopher C. Oberley is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Progenitor cell & Side population. The author has an hindex of 4, co-authored 4 publications receiving 62 citations. Previous affiliations of Christopher C. Oberley include University of Iowa.
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Journal ArticleDOI
Antioxidant proteins and reactive oxygen species are decreased in a murine epidermal side population with stem cell-like characteristics
Wanakee Carr,Rebecca E. Oberley-Deegan,Yuping Zhang,Christopher C. Oberley,Larry W. Oberley,Martine Dunnwald +5 more
TL;DR: The data indicate ROS and antioxidant levels are decreased in stem-like EpSPs, which exhibited less mitochondrial area, fewer peroxisomes and produced lower levels of ROS than Non-SPs.
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Withaferin A disrupts ubiquitin-based NEMO reorganization induced by canonical NF-κB signaling.
Shawn S. Jackson,Christopher C. Oberley,Christopher Hooper,Kreg M. Grindle,Shelly M. Wuerzberger-Davis,Jared Wolff,Kevin McCool,Lixin Rui,Shigeki Miyamoto +8 more
TL;DR: It is found that Withaferin A is a robust inhibitor of canonical and constitutive NF-κB activities, leading to apoptosis of certain lymphoma lines and a novel type of IKK inhibitor which acts by disrupting NEMO reorganization into ubiquitin-based signaling structures in vivo.
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Murine epidermal side population possesses unique angiogenic properties
Christopher C. Oberley,Francoise A. Gourronc,Shinkai Hakimi,Margaret Riordan,Sarah Bronner,Chunhua Jiao,Martine Dunnwald +6 more
TL;DR: It is concluded that EpSPs possess uniqueAngiogenic properties and that these cells may be indirectly responsible for the angiogenic response previously observed in the authors' ischemic limb model.
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Genetic background determines if Stat5b suppresses or enhances murine hepatocarcinogenesis
TL;DR: These results provide the first example of a single gene behaving as both oncogene and tumor suppressor in a given tissue, depending only on the endogenous modifier alleles carried by different genetic backgrounds.