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Christopher Cardozo

Researcher at Icahn School of Medicine at Mount Sinai

Publications -  156
Citations -  4649

Christopher Cardozo is an academic researcher from Icahn School of Medicine at Mount Sinai. The author has contributed to research in topics: Spinal cord injury & Muscle atrophy. The author has an hindex of 35, co-authored 133 publications receiving 3952 citations. Previous affiliations of Christopher Cardozo include United States Department of Veterans Affairs & Veterans Health Administration.

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Journal ArticleDOI

Evidence for the presence of five distinct proteolytic components in the pituitary multicatalytic proteinase complex. Properties of two components cleaving bonds on the carboxyl side of branched chain and small neutral amino acids.

Marian Orlowski, +2 more
- 01 Feb 1993 - 
TL;DR: Evidence is reported for the presence in the MPC of two additional distinct components, neither of them capable of cleaving the three model substrates, and the designation "branched chain amino acid preferring" (BrAAP) is proposed for this component.
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PGC-1α Expression Decreases in the Alzheimer Disease Brain as a Function of Dementia

Weiping Qin, +6 more
- 01 Mar 2009 - 
TL;DR: Therapeutic preservation of neuronal PGC-1alpha expression promotes the nonamyloidogenic processing of amyloid precursor protein precluding the generation of ameloidogenic Abeta peptides.
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Myostatin inhibits osteoblastic differentiation by suppressing osteocyte-derived exosomal microRNA-218: A novel mechanism in muscle-bone communication.

Yiwen Qin, +10 more
- 30 Jun 2017 - 
TL;DR: In this article, the authors show that myostatin-modified exosomes produced by Ocy454 cells that had been pre-treated with miR-218 could be taken up by osteoblastic MC3T3 cells, resulting in a marked reduction of Runx2, a key regulator of osteocyte differentiation, and in decreased osteoblast differentiation via the down-regulation of Wnt signaling pathway.
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Bone and muscle loss after spinal cord injury: organ interactions

Weiping Qin, +4 more
- 01 Nov 2010 - 
TL;DR: Adiposity and marrow fat are increased after SCI with intriguing, though poorly understood, implications for the function of skeletal muscle and bone cells.
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The Ubiquitin-Proteasome System: Potential Therapeutic Targets for Alzheimer's Disease and Spinal Cord Injury.

Bing Gong, +4 more
- 26 Jan 2016 - 
TL;DR: Advances in understanding of the role of several proteins of the UPS in Alzheimer’s disease (AD) and functional recovery after spinal cord injury (SCI) are reviewed to identify attractive and exciting targets for potential, future therapeutic interventions.