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Christopher K. Zarins

Researcher at Stanford University

Publications -  441
Citations -  34483

Christopher K. Zarins is an academic researcher from Stanford University. The author has contributed to research in topics: Aneurysm & Abdominal aortic aneurysm. The author has an hindex of 83, co-authored 435 publications receiving 33182 citations. Previous affiliations of Christopher K. Zarins include Medtronic plc & National Institutes of Health.

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Compensatory Enlargement of Human Atherosclerotic Coronary Arteries

TL;DR: It is concluded that human coronary arteries enlarge in relation to plaque area and that functionally important lumen stenosis may be delayed until the lesion occupies 40 percent of the internal elastic lamina area.
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Pulsatile flow and atherosclerosis in the human carotid bifurcation. Positive correlation between plaque location and low oscillating shear stress.

TL;DR: These studies confirm earlier findings under steady flow conditions that plaques tend to form in areas of low, rather than high, shear stress, but indicate in addition that marked oscillations in the direction of wall shear may enhance atherogenesis.
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Reporting standards for endovascular aortic aneurysm repair

TL;DR: This work is presented for the first time to the Ad Hoc Committee for Standardized Reporting Practices in Vascular Surgery of The Society for Vascular surgery/American Association forVascular Surgery.
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Carotid bifurcation atherosclerosis. Quantitative correlation of plaque localization with flow velocity profiles and wall shear stress.

TL;DR: It is concluded that in the human carotid bifurcation, regions of moderate to high shear stress, where flow remains unidirectional and axially aligned, are relatively spared of intimal thickening.
Journal Article

Hemodynamics and atherosclerosis. Insights and perspectives gained from studies of human arteries.

TL;DR: Development of clinical techniques for relating hemodynamic and tensile properties to plaque location, stenosis, and composition should permit pathologists to provide new insights into the bases for the topographic and individual differences in plaque progression and outcome.