Christy Mecey

TL;DR: It is shown that coronatine, but not its two biosynthetic precursors, also promotes interaction between Arabidopsis COI1 and multiple JAZ proteins, and the critical role of positively charged amino acids in the Jas domain in mediating the JA-Ile/coronatine-dependent JAZ interaction with COI 1 is illustrated.

TL;DR: It is discovered that AtMIN7 is required for not only PTI, consistent with the notion that Pst DC3000 degrades At MIN7 to suppressPTI, but also ETI, and blocking pathogen degradation of trans-Golgi network/early endosome-associated AtMIN 7 is a critical part of the ETI mechanism to counter bacterial suppression of PTI.

TL;DR: In this paper, mutational analysis of the C termini (containing the conserved Jas motif) of two JAZ repressors, JAZ1 and JAZ9, was conducted.

TL;DR: Conservation of related motifs and their necessity for the function of type III effectors from plant pathogens indicates that disturbing host pathways by mimicking activated host G-proteins may be a virulence mechanism employed by plant pathogens as well.

TL;DR: In this article, a genetic screening was conducted to identify Arabidopsis mutants that are impaired specifically in the development of disease symptoms (leaf chlorosis and/or necrosis) after infection with the bacterial pathogen Pseudomonas syringae pv tomato (Pst) DC3000.