C
Claire E. Kendal-Wright
Researcher at University of Hawaii at Manoa
Publications - 14
Citations - 323
Claire E. Kendal-Wright is an academic researcher from University of Hawaii at Manoa. The author has contributed to research in topics: Amnion & Fetal membrane. The author has an hindex of 8, co-authored 11 publications receiving 266 citations. Previous affiliations of Claire E. Kendal-Wright include Chaminade University of Honolulu.
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Journal ArticleDOI
Visfatin/Pre-B cell colony-enhancing factor in amniotic fluid in normal pregnancy, spontaneous labor at term, preterm labor and prelabor rupture of membranes: an association with subclinical intrauterine infection in preterm parturition.
Shali Mazaki-Tovi,Roberto Romero,Juan Pedro Kusanovic,Offer Erez,Francesca Gotsch,Pooja Mittal,Nandor Gabor Than,Chia Lang Nhan-Chang,Neil Hamill,Edi Vaisbuch,Tinnakorn Chaiworapongsa,Samuel S. Edwin,Jyh Kae Nien,Ricardo Gomez,Jimmy Espinoza,Claire E. Kendal-Wright,Sonia S. Hassan,Gillian D. Bryant-Greenwood +17 more
TL;DR: Visfatin is a physiologic constituent of AF and the concentration of AF visfatin increases with advancing gestational age, suggesting that visFatin participates in the host response against infection.
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Chronic Stretching of Amniotic Epithelial Cells Increases Pre-B Cell Colony-Enhancing Factor (PBEF/Visfatin) Expression and Protects Them from Apoptosis
TL;DR: Results show that chronic stretching of the amniotic epithelial cells increases PBEF expression, which protects them from apoptosis, and knocking-down PBEf with antisense probes abrogated this protective effect.
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Stretching, Mechanotransduction, and Proinflammatory Cytokines in the Fetal Membranes
TL;DR: It is proposed here that PBEF functions in normal pregnancy to protect the amnion cells as they become increasingly stretched, but if stimulated, it can initiate key events leading to parturition.
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Human amnion mesenchymal cells are pro-inflammatory when activated by the Toll-like receptor 2/6 ligand, macrophage-activating lipoprotein-2.
Brittany L.M. Sato,Eric S. Collier,Sheryl Anne D. Vermudez,Anthony D. Junker,Claire E. Kendal-Wright,Claire E. Kendal-Wright +5 more
TL;DR: The results suggest that AMC elicit a pro-inflammatory response following stimulation with the known TLR2/6 ligand MALP-2, and supports the idea that AMC express the innate immune system receptors that could help with immune surveillance during infection and contribute to inflammatory responses that lead to PPROM.
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The Role of Danger Associated Molecular Patterns in Human Fetal Membrane Weakening
TL;DR: The knowledge to date pertinent to the role of DAMPs and PRRs in fetal membrane weakening is summarized and the clinical potential brought by a better understanding of these pathways by pathway manipulation strategies is discussed.