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Claudio Gentili

Researcher at University of Padua

Publications -  121
Citations -  4014

Claudio Gentili is an academic researcher from University of Padua. The author has contributed to research in topics: Mood & Anxiety. The author has an hindex of 26, co-authored 116 publications receiving 3161 citations. Previous affiliations of Claudio Gentili include University of Strasbourg & IMT Institute for Advanced Studies Lucca.

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Efficacy of Psychotherapies for Borderline Personality Disorder: A Systematic Review and Meta-analysis

TL;DR: Psychotherapies, most notably dialectical behavior therapy and psychodynamic approaches, are effective for borderline symptoms and related problems but effects are small, inflated by risk of bias and publication bias, and particularly unstable at follow-up.
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Selective aberrant functional connectivity of resting state networks in social anxiety disorder.

TL;DR: A relationship between functional connectivity and disease severity was found in specific regions of RSNs, including medial and lateral prefrontal cortex, as well as parietal and occipital regions.
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The Effect of Visual Experience on the Development of Functional Architecture in hMT

TL;DR: Results demonstrate that activation of h MT+ and surrounding cortex by tactile flow is not mediated by visual mental imagery and that the functional organization of hMT+ can develop to subserve tactile flow perception in the absence of any visual experience.
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Neural correlates of spatial working memory in humans: A functional magnetic resonance imaging study comparing visual and tactile processes

TL;DR: These findings provide a neurobiological support to behavioral observations by indicating that common cerebral regions subserve generation of higher order mental representations involved in working memory independently from a specific sensory modality.
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Altered Effective Connectivity Network of the Amygdala in Social Anxiety Disorder: A Resting-State fMRI Study

TL;DR: This study is the first to reveal a network of abnormal effective connectivity of core structures in SAD and lends neurobiological support towards cognitive models considering disinhibition and an attentional bias towards negative stimuli as a core feature of the disorder.