Clémence Mille

TL;DR: The observation of a CpG-specific, locus-specific methylation pattern, although it can provide an epigenetic biomarker of a multifactorial disease, does not indicate whether the reported epigenetic pattern preexists or follows the establishment of T1D.

TL;DR: The current results suggest that the methylation of IGF1 CpG-137 contributes to the individual variation of fetal growth by regulating IGF1 expression in fetal tissues.

TL;DR: The DMR identified in this study presents suggestive evidence for altered methylation site in T1D and provides a promising new candidate gene, and suggests that hypo‐methylation could increase the fetal exposure to environmental factors in T 1D susceptibility.

TL;DR: After publication of the original article, it came to the publishers’ attention that the below author’s corrections provided at the proofing stage had been misinterpreted.

TL;DR: The application of genetic code expansion in combination with photo-crosslinking chemistry, as well as "optoproteomics", to identify proteins interacting with the human L-type membrane amino acid transporter 3 (LAT3), validating the approach and providing insights into the LAT3 protein-protein interaction network currently unavailable.