C
Clementina Bianchi
Researcher at University of Pisa
Publications - 14
Citations - 478
Clementina Bianchi is an academic researcher from University of Pisa. The author has contributed to research in topics: Acetylcholine & Cholinergic. The author has an hindex of 10, co-authored 14 publications receiving 474 citations. Previous affiliations of Clementina Bianchi include University of Chieti-Pescara.
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The cerebral acetylcholine release in conscious rabbits with semi-permanently implanted epidural cups.
TL;DR: This method was employed to examine the relationship between behaviour, electrocortical activity and cortical acetylcholine output in conscious, freely moving animals to assess the impact of drugs on the activity of cholinergic pathways belonging to the reticulocortical ascending system.
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The effect of catecholamines and sympathetic stimulation on the release of acetylcholine from the guinea-pig colon
TL;DR: It is suggested that the sympathetic control of gastrointestinal tone and motility is exerted through two different routes: inhibition of intramural cholinergic plexuses and direct relaxation of smooth muscle cells.
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Effect of amantadine on cerebral acetyl-choline release and content in the guinea pig
L. Beani,Clementina Bianchi +1 more
TL;DR: The conclusion is reached that the role of dopamine in determining this counter-action is predominant and is based on the hypothesis that the activation of the cerebral adrenergic neurons is associated with a cholinergic counter- action.
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Effects of metoclopramide on isolated guinea-pig colon. 2. Interference with ganglionic stimulant drugs.
TL;DR: It is suggested that the drug blocks tryptaminergic receptors, necessary for activating unknown nervous elements involved in the control of gastrointestinal tone and motility in isolated guinea-pig distal colon.
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Effects of metoclopramide on isolated guinea-pig colon. 1)Peripheral sensitization to acetylcholine
TL;DR: Interestingly, morphine, but not hexamethonium, counteracted effects of metoclopramide suggesting interference of the drug on non-adrenergic, non-cholinergic intramural nervous structures, in addition to the demonstrated peripheral sensitization of muscarinic receptors.