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D. A. J. Tyrrell
Researcher at Salisbury University
Publications - 44
Citations - 3858
D. A. J. Tyrrell is an academic researcher from Salisbury University. The author has contributed to research in topics: Rhinovirus & Common cold. The author has an hindex of 24, co-authored 44 publications receiving 3682 citations. Previous affiliations of D. A. J. Tyrrell include Cardiff University & Medical Research Council.
Papers
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Journal ArticleDOI
Psychological Stress and Susceptibility to the Common Cold
TL;DR: Psychological stress was associated in a dose-response manner with an increased risk of acute infectious respiratory illness, and this risk was attributable to increased rates of infection rather than to an increased frequency of symptoms after infection.
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Negative life events, perceived stress, negative affect, and susceptibility to the common cold
TL;DR: This paper found that higher scores on each of the three stress scales were associated with a greater risk of developing a cold, but the relation between stressful life events and illness was mediated by a different biologic process than were relations between perceived stress and illness and negative affect and illness.
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Smoking, alcohol consumption, and susceptibility to the common cold.
TL;DR: Smokers were at greater risk for developing colds than nonsmokers because smokers were more likely both to develop infections and to develop illness following infection, and moderate alcohol consumption was associated with decreased risk for nonsmoker.
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Signs and symptoms in common colds.
TL;DR: The results confirm indication from earlier studies that the main difference between colds induced by different viruses is in duration of the incubation period and justify treatment with different drugs either successively or simultaneously.
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Circulating tumour necrosis factor-alpha and interferon-gamma are detectable during acute and convalescent parvovirus B19 infection and are associated with prolonged and chronic fatigue.
Jonathan R. Kerr,Faraj Barah,Derek L. Mattey,Ian Laing,Stephen J. Hopkins,Ian V. Hutchinson,D. A. J. Tyrrell +6 more
TL;DR: Prolonged upregulation of serum IFN-gamma and TNF-alpha appears to represent a consistent host response to symptomatic B19 virus infection.