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Dana Egozi

Researcher at Kaplan Medical Center

Publications -  38
Citations -  1011

Dana Egozi is an academic researcher from Kaplan Medical Center. The author has contributed to research in topics: Medicine & Controlled release. The author has an hindex of 16, co-authored 34 publications receiving 862 citations. Previous affiliations of Dana Egozi include Technion – Israel Institute of Technology & Rambam Health Care Campus.

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Improved vascular organization enhances functional integration of engineered skeletal muscle grafts.

TL;DR: A “relay” approach is suggested in which extended in vitro incubation, enabling the formation of a more structured vascular bed, allows for graft-host angiogenic collaboration that promotes anastomosis and vascular integration that supports enhanced muscle regeneration, maturation, and integration.
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An engineered muscle flap for reconstruction of large soft tissue defects

TL;DR: The described engineered muscle flap, equipped with an autologous vascular pedicle, constitutes an effective tool for reconstruction of large defects, thereby circumventing the need for both harvesting autologously flaps and postoperative scarification.
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Adipose-derived endothelial and mesenchymal stem cells enhance vascular network formation on three-dimensional constructs in vitro

TL;DR: These data demonstrate optimal vascular network formation upon co-culture of microvascular endothelial cells and adipose-derived MSCs in vitro and constitute a significant step in appreciation of the potential of microVascular endothelium cells and M SCs in different tissue engineering applications that can also be advantageous in in vivo studies.
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Erythropoietin improves the survival of fat tissue after its transplantation in nude mice.

TL;DR: The data suggest that stimulation of angiogenesis by a cluster ofAngiogenic factors and decreased fat cell apoptosis account for potential mechanisms that underlie the improved long-term survival of fat transplants following EPO treatment.
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Fibronectin potentiates topical erythropoietin-induced wound repair in diabetic mice.

TL;DR: It is shown that EPO and FN have an additive effect on wound repair in diabetic mice and this effect was associated with increased angiogenesis, increased eNOS and β1-integrin expression, and reduced expression of inflammatory cytokines and apoptosis.