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Daniela Tagliasacchi

Researcher at University of Modena and Reggio Emilia

Publications -  11
Citations -  669

Daniela Tagliasacchi is an academic researcher from University of Modena and Reggio Emilia. The author has contributed to research in topics: Live birth & Aromatase. The author has an hindex of 8, co-authored 10 publications receiving 571 citations.

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LH and hCG action on the same receptor results in quantitatively and qualitatively different intracellular signalling.

TL;DR: It is concluded that the LHCGR is able to differentiate the activity of hLH and hCG and respond to equipotent, constant hH or hCG stimulation with a fluctuating cAMP production and progressive progesterone secretion.
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Anti-Müllerian hormone-based prediction model for a live birth in assisted reproduction

TL;DR: The success of IVF was found to mainly depend on maternal age and serum AMH concentrations, one of the most relevant and valuable markers of ovarian reserve.
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Comparison of a gonadotropin-releasing hormone (GnRH) antagonist and GnRH agonist flare-up regimen in poor responders undergoing ovarian stimulation

TL;DR: The flare-up protocol appears to be more effective than the GnRH-antagonist protocol in terms of mature oocytes retrieved, fertilization rate, and top-quality embryos transferred in poor-responder patients.
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Live birth rates in the different combinations of the Bologna criteria poor ovarian responders: a validation study

TL;DR: The study demonstrates that the different groups of poor responders based on the Bologna criteria have similar IVF outcomes and proves that the BOLOGna criteria poor responders in the various subgroups represent a homogenous population with similar pre-clinical and clinical outcomes.
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The anti-Müllerian hormone (AMH) acts as a gatekeeper of ovarian steroidogenesis inhibiting the granulosa cell response to both FSH and LH

TL;DR: These findings contribute in clarifying the relationship between hormones regulating the early phase of steroidogenesis confirming that AMH is playing a suppressive role on CYP19A1 expression stimulated by gonadotropin in hGCs.