D
Danmei Li
Researcher at University of California, San Diego
Publications - 12
Citations - 585
Danmei Li is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Gonadotropic cell & Growth factor. The author has an hindex of 8, co-authored 12 publications receiving 563 citations.
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Journal ArticleDOI
A functional bone morphogenetic protein system in the ovary
Shunichi Shimasaki,R. J. Zachow,Danmei Li,H. Kim,Shun-ichiro Iemura,Naoto Ueno,Karuna Sampath,R. J. Chang,Gregory F. Erickson +8 more
TL;DR: Bone morphogenetic proteins have now been identified as molecules that differentially regulate FSH-dependent estradiol and progesterone production in a way that reflects steroidogenesis during the normal estrous cycle, and it can be hypothesized that BMPs might be the long-sought "luteinization inhibitor" in Graafian follicles during their growth and development.
Journal ArticleDOI
Mechanism of selective retinoid X receptor agonist-induced hypothyroidism in the rat.
Sha Liu,Kathleen M. Ogilvie,Kay Klausing,Mark A. Lawson,Diane Jolley,Danmei Li,James M. Bilakovics,Bernadette Pascual,Nancy I. Hein,Mary S. Urcan,Mark D. Leibowitz +10 more
TL;DR: An unexpected mechanism by which rexinoids induce hypothyroidism by acutely reducing TSH secretion from the anterior pituitary is identified, independent of the rex inoid's previously demonstrated inhibition of TSHbeta gene transcription.
Journal ArticleDOI
Extrapituitary actions of gonadotropin-releasing hormone: stimulation of insulin-like growth factor-binding protein-4 and atresia.
Gregory F. Erickson,Danmei Li,Rajabali Sadrkhanloo,Xin-Jun Liu,Shunichi Shimasaki,Nicholas Ling +5 more
TL;DR: The hypothesis that autocrine/paracrine secretion of ovarian GnRH might cause atresia by mechanisms involving increased IGFBP-4 synthesis is supported.
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Endogenous insulin-like growth factor-I is obligatory for stimulation of rat inhibin alpha-subunit expression by follicle-stimulating hormone.
TL;DR: It is proposed that the IGF-I signaling is obligatory for FSH stimulation of Inh-alpha expression in rat GC and requires activation of protein tyrosine kinases by endogenously produced IGF-i.
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Gonadotropin-releasing hormone overcomes follicle-stimulating hormone's inhibition of insulin-like growth factor-5 synthesis and promotion of its degradation in rat granulosa cells
TL;DR: The effect of a gonadotropin-releasing hormone-agonist (GnRH-a) on the synthesis of insulin-like growth factor-binding protein-5 (IGFBP-5), a physiological marker for atresia, was investigated and IGF BP-5 mRNA accumulation levels were increased by Gn RH-a in a dose dependent manner.