scispace - formally typeset
D

David Broom

Researcher at Coventry University

Publications -  80
Citations -  1893

David Broom is an academic researcher from Coventry University. The author has contributed to research in topics: Medicine & Aerobic exercise. The author has an hindex of 17, co-authored 61 publications receiving 1375 citations. Previous affiliations of David Broom include Sheffield Hallam University & Leeds Beckett University.

Papers
More filters
Journal ArticleDOI

Influence of resistance and aerobic exercise on hunger, circulating levels of acylated ghrelin, and peptide YY in healthy males.

TL;DR: The findings suggest ghrelin and PYY may regulate appetite during and after exercise, but further research is required to establish whether exercise-induced changes in ghrel in and peptide YY influence subsequent food intake.
Journal ArticleDOI

Exercise-induced suppression of acylated ghrelin in humans

TL;DR: It is demonstrated that plasma acylated ghrelin concentration and hunger are suppressed during running.
Journal ArticleDOI

Acute and Chronic Effects of Exercise on Appetite, Energy Intake, and Appetite-Related Hormones: The Modulating Effect of Adiposity, Sex, and Habitual Physical Activity.

TL;DR: The balance of evidence suggests that adiposity and sex do not modify appetite or energy intake responses to acute or chronic exercise interventions, but individuals with higher habitual physical activity levels may better adjust energy intake in response to energy balance perturbations.
Journal ArticleDOI

Barriers and motivators of physical activity participation in middle-aged and older-adults – a systematic review

TL;DR: Barriers are comparable across the two age groups with environmental factors and resources being the most commonly identified barriers and social influences were most important.
Journal ArticleDOI

A single session of treadmill running has no effect on plasma total ghrelin concentrations.

TL;DR: Findings indicate that treadmill running suppresses hunger but this effect is not mediated by changes in plasma total ghrelin concentration.