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Denis C. Muller

Researcher at National Institutes of Health

Publications -  91
Citations -  11612

Denis C. Muller is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Body mass index & Insulin. The author has an hindex of 55, co-authored 91 publications receiving 11059 citations. Previous affiliations of Denis C. Muller include University of Maryland, Baltimore & Johns Hopkins University.

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Predictors of Progression From Impaired Glucose Tolerance to NIDDM: An Analysis of Six Prospective Studies

TL;DR: Analysis of data from six prospective studies indicates that persons with IGT are at high risk of NIDDM and that further refinement of risk can be made by other simple measurements, and the ability to identify persons at high Risk should facilitate clinical trials in diabetes prevention.
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Metabolic Syndrome in Men With Prostate Cancer Undergoing Long-Term Androgen-Deprivation Therapy

TL;DR: Data suggest that metabolic syndrome was present in more than 50% of the men undergoing long-term ADT, predisposing them to higher cardiovascular risk.
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Dietary patterns and changes in body mass index and waist circumference in adults

TL;DR: Consuming a diet high in fruit, vegetables, reduced-fat dairy, and whole grains and low in red and processed meat, fast food, and soda was associated with smaller gains in BMI and waist circumference.
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Pulse wave velocity is an independent predictor of the longitudinal increase in systolic blood pressure and of incident hypertension in the Baltimore Longitudinal Study of Aging.

TL;DR: PWV could help identify normotensive individuals who should be targeted for the implementation of interventions aimed at preventing or delaying the progression of subclinical arterial stiffening and the onset of hypertension.
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Metabolic syndrome amplifies the age-associated increases in vascular thickness and stiffness

TL;DR: It is suggested that the components of MS interact to synergistically impact vascular thickness and stiffness and that the excess cardiovascular risk associated with MS is partly mediated through the amplified alterations in these vascular properties.