D
Dianne B. McKay
Researcher at University of California, San Diego
Publications - 65
Citations - 3722
Dianne B. McKay is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Transplantation & Innate immune system. The author has an hindex of 27, co-authored 64 publications receiving 3300 citations. Previous affiliations of Dianne B. McKay include Harvard University & Sharp Memorial Hospital.
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Journal ArticleDOI
Inflammation in AKI: Current Understanding, Key Questions, and Knowledge Gaps
Hamid Rabb,Matthew D. Griffin,Dianne B. McKay,Sundararaman Swaminathan,Peter Pickkers,Mitchell H. Rosner,John A. Kellum,Claudio Ronco +7 more
TL;DR: The most important recent developments in understanding the inflammatory mechanisms of AKI are summarized, key limitations of the commonly used animal models and clinical trial designs that may prevent successful clinical application are highlighted, and priority approaches for research toward clinical translation are suggested.
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Reproduction and Transplantation: Report on the AST Consensus Conference on Reproductive Issues and Transplantation
TL;DR: This study highlights the need to understand more fully the pre- and post-transplant role of immune suppression in the selection and timing ofitoneal organ transplants.
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Pregnancy in Recipients of Solid Organs — Effects on Mother and Child
TL;DR: The most recent data about the effects on the mother and child of pregnancy in the recipients of solid-organ transplants are discussed.
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Nonadherence consensus conference summary report.
Richard N. Fine,Yolanda T. Becker,S. De Geest,Howard J. Eisen,R. Ettenger,R. Evans,D. Lapointe Rudow,Dianne B. McKay,Alicia M. Neu,Thomas E. Nevins,Jorge Reyes,Jo Wray,Fabienne Dobbels +12 more
TL;DR: It was found that nonadherence to immunosuppressants is more prevalent than the authors assume and hard to measure accurately, and some roadmaps for future studies on this complicated, multifaceted problem are provided.
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TLR2 Is Constitutively Expressed within the Kidney and Participates in Ischemic Renal Injury through Both MyD88-Dependent and -Independent Pathways
Alana A. Shigeoka,Todd Holscher,Andrew J. King,Frank W. Hall,William B. Kiosses,Peter S. Tobias,Nigel Mackman,Dianne B. McKay,Dianne B. McKay +8 more
TL;DR: It is concluded that TLR2 protein is constitutively expressed in the kidney and plays an important role in the pathogenesis of acute ischemic injury by signaling both MyD88-dependent and MyD 88-independent pathways.