D
Dirk H. Hellhammer
Researcher at University of Trier
Publications - 178
Citations - 41595
Dirk H. Hellhammer is an academic researcher from University of Trier. The author has contributed to research in topics: Trier social stress test & Cortisol awakening response. The author has an hindex of 80, co-authored 178 publications receiving 38769 citations. Previous affiliations of Dirk H. Hellhammer include University of Mainz & University of Southampton.
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The ‘Trier Social Stress Test’ – A Tool for Investigating Psychobiological Stress Responses in a Laboratory Setting
TL;DR: The results suggest that gender, genetics and nicotine consumption can influence the individual's stress responsiveness to psychological stress while personality traits showed no correlation with cortisol responses to TSST stimulation.
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Two formulas for computation of the area under the curve represent measures of total hormone concentration versus time-dependent change
TL;DR: It is shown that depending on which formula is used, different associations with other variables may emerge, and it is recommended to employ both formulas when analyzing data sets with repeated measures.
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Salivary cortisol in psychoneuroendocrine research: Recent developments and applications
TL;DR: An up-to-date overview of recent methodological developments, novel applications as well as a discussion of possible future applications of salivary cortisol determination are provided.
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Impact of gender, menstrual cycle phase, and oral contraceptives on the activity of the hypothalamus-pituitary-adrenal axis.
TL;DR: Although men seem to have a stronger hypothalamic drive in response to stressful stimulation than women, differences in salivary-free cortisol levels, at least in part, may be explained by estradiol-induced changes in corticosteroid-binding protein levels.
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The potential role of hypocortisolism in the pathophysiology of stress-related bodily disorders.
TL;DR: It is proposed that a persistent lack of cortisol availability in traumatized or chronically stressed individuals may promote an increased vulnerability for the development of stress-related bodily disorders.