D
Do-Hee Kim
Researcher at Kyonggi University
Publications - 146
Citations - 3335
Do-Hee Kim is an academic researcher from Kyonggi University. The author has contributed to research in topics: Apoptosis & Phosphorylation. The author has an hindex of 30, co-authored 125 publications receiving 2559 citations. Previous affiliations of Do-Hee Kim include Seoul National University & New Generation University College.
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Journal ArticleDOI
Ghrelin stimulates proliferation and differentiation and inhibits apoptosis in osteoblastic MC3T3-E1 cells
Sang Wan Kim,Sun Ju Her,Seong Jae Park,Do-Hee Kim,Do-Hee Kim,Kyong Soo Park,Kyong Soo Park,Hong Kyu Lee,Hong Kyu Lee,Byung Hee Han,Min Seon Kim,Chan Soo Shin,Chan Soo Shin,Seong Yeon Kim,Seong Yeon Kim +14 more
TL;DR: Results suggest that ghrelin promotes proliferation and differentiation and inhibits apoptosis of osteoblasts and enhanced in vitro osteoblast differentiation as evidenced by matrix mineralization, alkaline phosphatase activity, and osteOBlast-specific gene expression.
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The Role of Vitamin D in Thyroid Diseases
TL;DR: This review will focus on recent data on the possible role of vitamin D in thyroid diseases, including autoimmune thyroid diseases and thyroid cancers.
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A novel adipokine CTRP1 stimulates aldosterone production
Jun Ho Jeon,Kun-yong Kim,Jae Hyeong Kim,Ahmi Baek,Hyungin Cho,Young Ho Lee,Jong-Wan Kim,Do-Hee Kim,Seung Hyun Han,Jong-Seok Lim,Keun Il Kim,Do Young Yoon,Soo Hyun Kim,Goo Taeg Oh,Eunjoon Kim,Young Yang +15 more
TL;DR: It was revealed that angiotensin II‐induced aldosterone production is, at least in part, mediated by the stimulation of CTRp1 secretion, not by the increase of CTRP1 mRNA transcription.
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Nitric oxide activates Nrf2 through S-nitrosylation of Keap1 in PC12 cells.
TL;DR: Investigating whether the cytoprotective effect of NO is mediated through Nrf2 activation by directly modifying cysteine residues of Keap1 reveals that treatment of rat pheochromocytoma cells with an NO donor S-nitroso-N-acetylpenicillamine (SNAP) induced nuclear translocation and DNA binding of NRF2.
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Diallyl trisulfide induces apoptosis in human breast cancer cells through ROS-mediated activation of JNK and AP-1
TL;DR: Oral administration of 5μmol/kg DATS to female Balb/c mice inhibited the growth of human MCF-7 cell tumor xenografts and suggest that DATS-induced apoptosis is mediated through ROS generation and subsequent activation of JNK and AP-1.